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Study of gastroepiploic conductance arteries and microvessels revealed that the contribution of EDHF to endothelium-dependent relaxations is significantly larger in human microvessels than in large arteries, that EDHF-mediated relaxations are mediated via activation of K+ channels, and that risk factors, particularly hypercholesterolemia and aging, were associated with impaired EDHF-mediated dilat
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