<http://webisa.webdatacommons.org/160303694>	<http://www.w3.org/ns/prov#wasQuotedFrom>	<nih.gov> .
<http://webisa.webdatacommons.org/160303694>	<http://www.w3.org/1999/02/22-rdf-syntax-ns#type>	<http://www.w3.org/ns/prov#Entity> .
<http://webisa.webdatacommons.org/prov/160234552>	<http://www.w3.org/ns/prov#wasDerivedFrom>	<http://webisa.webdatacommons.org/160303694> .
<http://webisa.webdatacommons.org/prov/273012291>	<http://www.w3.org/ns/prov#wasDerivedFrom>	<http://webisa.webdatacommons.org/160303694> .
<http://webisa.webdatacommons.org/160303694>	<http://www.w3.org/ns/prov#value>	"Study of gastroepiploic conductance arteries and microvessels revealed that the contribution of EDHF to endothelium-dependent relaxations is significantly larger in human microvessels than in large arteries, that EDHF-mediated relaxations are mediated via activation of K+ channels, and that risk factors, particularly hypercholesterolemia and aging, were associated with impaired EDHF-mediated dilat" .