. . "In addition, aminoguanidine decreases experimentally-induced ischemic infarct volume in the absence of exogenous AGEs, suggesting a potential therapy for stroke and related maladies involving or including ischemia or reperfusion injury, that damage brain, heart, bowel or other organs or tissues, including as examples, peripheral vascular occlusion injury, diabetic retinopathy and damage attendant" . . . .