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  • Indeed it appears that that the primary proteins implicated in these diseases, including A??, tau, ??-synuclein, huntingtin, TDP-43 and PrP, are members of the innate immune system and their activity and assembly into oligomers and amyloids are not protein misfolding events or prion activities, as proposed by the protein misfolding concept and the prion hypothesis, but part of their repertoire of
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