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  • In addition to the claim that the prion hypothesis and protein misfolding theory are flawed, I proposed that the primary proteins implicated in these diseases, including A??, tau, ??-synuclein, huntingtin, TDP-43 and PrP, are members of the innate immune system and their activity and assembly into oligomers and amyloids are not protein misfolding events or prion activities, as proposed by the prot
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