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  • Low-density lipoprotein cholesterol (LDL-C) particles penetrate the outer layer of the vessel wall (endothelial lining), become oxidized, and form foam cells when engulfed by macrophages.11 Foam cells, free lipids, inflammatory cells, and immune cells accumulate to form an atheroma.12 A variety of patient-specific characteristics facilitate the process, such as hypertension and smoking.
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